{"id":760426,"date":"2019-01-08T12:40:44","date_gmt":"2019-01-08T10:40:44","guid":{"rendered":"https:\/\/homomedical.com\/formaciones\/programa-de-formacion-de-especialidad-endocrinologia\/"},"modified":"2022-11-21T19:05:05","modified_gmt":"2022-11-21T17:05:05","slug":"endocrinology-specialization-training-program-for-european-resident-physicians","status":"publish","type":"product","link":"https:\/\/pyreneesworldtravels.com\/en\/formaciones\/endocrinology-specialization-training-program-for-european-resident-physicians\/","title":{"rendered":"Endocrinology Specialization Training Program (For European Resident Physicians"},"content":{"rendered":"<table width=\"747\">\n<tbody>\n<tr>\n<td width=\"67\">YEAR<\/td>\n<td width=\"255\">RESIDENCY<\/td>\n<td width=\"111\">MONTHS<\/td>\n<td width=\"88\">WEEKS<\/td>\n<td width=\"227\">HOURS<\/td>\n<\/tr>\n<tr>\n<td width=\"67\">&nbsp;<\/p>\n<p>First<\/td>\n<td width=\"255\">Internal medicine<\/p>\n<p>&nbsp;<\/p>\n<p>Endocrinology service for adults<\/td>\n<td width=\"111\">3*<\/p>\n<p>&nbsp;<\/p>\n<p>8<\/td>\n<td width=\"88\">13<\/p>\n<p>&nbsp;<\/p>\n<p>35<\/td>\n<td width=\"227\">60 hours per week**<\/p>\n<p>&nbsp;<\/p>\n<p>60 hours per week<\/td>\n<\/tr>\n<tr>\n<td width=\"67\">&nbsp;<\/p>\n<p>&nbsp;<\/p>\n<p>Second<\/td>\n<td width=\"255\">General endocrinology<\/p>\n<p>Endocrinology and pregnancy<\/p>\n<p>&nbsp;<\/p>\n<p>Center for education and care of the diabetic patient<\/p>\n<p>Endocrinology laboratory<\/td>\n<td width=\"111\">6<\/p>\n<p>2<\/p>\n<p>&nbsp;<\/p>\n<p>1<\/p>\n<p>2<\/td>\n<td width=\"88\">24<\/p>\n<p>8<\/p>\n<p>&nbsp;<\/p>\n<p>4<\/p>\n<p>8<\/td>\n<td width=\"227\">60 hours per week<\/p>\n<p>60 hours per week<\/p>\n<p>&nbsp;<\/p>\n<p>60 hours per week<\/p>\n<p>60 hours per week<\/td>\n<\/tr>\n<tr>\n<td width=\"67\">&nbsp;<\/p>\n<p>Third<\/td>\n<td width=\"255\">General endocrinology<\/p>\n<p>Pediatric endocrinology service<\/td>\n<td width=\"111\">8<\/p>\n<p>&nbsp;<\/p>\n<p>3<\/td>\n<td width=\"88\">32<\/p>\n<p>&nbsp;<\/p>\n<p>12<\/td>\n<td width=\"227\">60 hours per week<\/p>\n<p>&nbsp;<\/p>\n<p>60 hours per week<\/td>\n<\/tr>\n<\/tbody>\n<\/table>\n<ol>\n<li><strong> Diseases and syndromes treated in intermediate therapy services and clinical rooms<\/strong><\/li>\n<li>Acute bronchial asthma crisis and severe acute asthma<\/li>\n<\/ol>\n<p>Adult respiratory failure<\/p>\n<ol start=\"2\">\n<li>Acute myocardial infarction<\/li>\n<li>Hypertensive crisis<\/li>\n<li>Arrhythmias<\/li>\n<li>Chest pain<\/li>\n<li>Heart failure and acute pulmonary edema<\/li>\n<li>Cerebrovascular disease<\/li>\n<li>Cardiorespiratory arrest<\/li>\n<li>Pulmonary thromboembolism and hypertensive pulmonary heart disease<\/li>\n<li>Acute abdominal pain<\/li>\n<li>Follow-up in the case of acute diarrhea<\/li>\n<li>Follow-up in the case of anuria<\/li>\n<li>Altered hydromineral balance<\/li>\n<li>Meningoencephalitis<\/li>\n<li>Acute febrile illness<\/li>\n<li>Upper and lower gastrointestinal bleeding<\/li>\n<li>Shock<\/li>\n<li>Acute poisoning<\/li>\n<li>Endocrine emergencies:<\/li>\n<\/ol>\n<ul>\n<li>Ketosis and diabetic ketoacidosis<\/li>\n<li>Lactic acidosis<\/li>\n<li>Nonketotic hyperosmolar coma<\/li>\n<li>Hypoglycemia<\/li>\n<li>Adrenal crisis<\/li>\n<li>Myxedema coma<\/li>\n<li>Thyroid crisis<\/li>\n<li>Acute hypocalcemia<\/li>\n<\/ul>\n<ol start=\"20\">\n<li>Prevention and control of hospital-acquired diseases<\/li>\n<li>Medical emergencies in pregnant women<\/li>\n<li>Ethical principles in the care of emergency patients and their families<\/li>\n<\/ol>\n<p>&nbsp;<\/p>\n<p>The following aspects are analyzed for each of the diseases studied:<\/p>\n<p>Concept, etiology, epidemiology, classification, clinical forms, pathophysiology, clinical manifestations, complementary tests, diagnosis, differential diagnosis, evolution, complications, sequelae, prognosis, comprehensive treatment (prophylactic, medical-surgical, acute and supportive), rehabilitation<\/p>\n<ol>\n<li><strong> Basic principles in the endocrinology laboratory<\/strong><\/li>\n<li>Radioimmunoassay; hormones, proteins and steroids; ultra-microanalytical system<\/li>\n<li>Fundamentals, indications, procedures and interpretation of baseline measurements or dynamic tests<\/li>\n<li>Diagnostic procedures on: hypothalamus, pituitary gland, thyroid, parathyroid, pancreas and lipids, adrenal cortex, adrenal medulla, ovaries, testicles<\/li>\n<li>Female reproduction, cytogenetics and immunology laboratories<\/li>\n<\/ol>\n<p><strong>III. Introductory clinical endocrinology, CH, embryology, anatomy, histology and physiology of the endocrine system:<\/strong><\/p>\n<ol>\n<li><strong><u>General concepts of endocrinology<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept of endocrine regulation<\/li>\n<li>Feedforward<\/li>\n<li>Feedback. Positive and negative feedback<\/li>\n<li>Hormonal and humoral feedback<\/li>\n<li>Long, short and ultra-short loop feedback<\/li>\n<li>Hormonal biosynthesis patterns<\/li>\n<li>Definition of receptors and their cellular location<\/li>\n<li>General mechanisms of hormone action<\/li>\n<\/ul>\n<ol start=\"2\">\n<li><strong><u>Neuroendocrinology<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Cortex-endocrine gland relationship<\/li>\n<li>Neurotransmitters<\/li>\n<li>Cortico-hypothalamus-pituitary relationship<\/li>\n<\/ul>\n<ol start=\"3\">\n<li><strong><u>Adeno and neurohypophysis<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Embryology<\/li>\n<li>Normal histology<\/li>\n<li>Anatomy<\/li>\n<li>Physiology<\/li>\n<li>Hypothalamic-pituitary relationships<\/li>\n<li>Peripheral actions of pituitary hormones and mechanisms of action<\/li>\n<\/ul>\n<ol start=\"4\">\n<li><strong><u>Thyroid<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Embryology<\/li>\n<li>Normal histology<\/li>\n<li>Anatomical relationships<\/li>\n<li>Iodine kinetics<\/li>\n<li>Hypothalamus-pituitary-thyroid regulation<\/li>\n<li>Thyroid autoregulation<\/li>\n<li>Thyroid hormone synthesis<\/li>\n<li>Peripheral actions of thyroid hormones and mechanism of their action<\/li>\n<\/ul>\n<ol start=\"5\">\n<li><strong><u>Parathyroid<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Embryology<\/li>\n<li>Histology<\/li>\n<li>Anatomy<\/li>\n<li>Physiology<\/li>\n<li>Peripheral actions of PTH, calcitonin and vitamin D<\/li>\n<li>PTH synthesis and secretion<\/li>\n<li>Mineral metabolism (calcium, phosphorus and magnesium)<\/li>\n<li>Skeletal and mineral homeostasis<\/li>\n<\/ul>\n<ol start=\"6\">\n<li><strong><u>Pancreas<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Embryology<\/li>\n<li>Histology<\/li>\n<li>Anatomy<\/li>\n<li>Hormone production<\/li>\n<li>Insulin and glucagon<\/li>\n<li>Insulin secretion<\/li>\n<li>Insulin mechanism of action<\/li>\n<li>Regulation of insulin synthesis\n<ul>\n<li>Glucagon actions<\/li>\n<li>Other counterregulatory hormones and their action<\/li>\n<li>Fed and fasted glucose homeostasis<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<ol start=\"7\">\n<li><strong><u>Lipid metabolism<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Triglyceride metabolism: fat for energy<\/li>\n<li>Cholesterol metabolism<\/li>\n<li>Hyperlipidemia<\/li>\n<li>Atherosclerosis<\/li>\n<\/ul>\n<ol start=\"8\">\n<li><strong><u>Adrenal cortex and medulla<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Embryology<\/li>\n<li>Histology<\/li>\n<li>Anatomy<\/li>\n<li>Steroidogenesis<\/li>\n<li>Hypothalamic-pituitary-adrenal axis<\/li>\n<\/ul>\n<ul>\n<li>Renin-angiotensin-aldosterone axis<\/li>\n<li>Circadian rhythm<\/li>\n<li>Peripheral actions of mineralocorticoids and glucocorticoids<\/li>\n<li>Hormonal mechanism of action of steroid hormones<\/li>\n<li>Biosynthesis of catecholamines<\/li>\n<li>Actions of catecholamines<\/li>\n<li>APUD system\n<ol>\n<li><strong><u>Testicles<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Embryology<\/li>\n<li>Histology<\/li>\n<li>Anatomy<\/li>\n<li>Testicular physiology<\/li>\n<li>Hypothalamic-pituitary-testicular axis<\/li>\n<li>Spermatogenesis<\/li>\n<li>Peripheral actions of testosterone and dihydrotestosterone<\/li>\n<li>Mechanism of action of androgens<\/li>\n<li>Importance of Sertoli cells<\/li>\n<\/ul>\n<ol start=\"10\">\n<li><strong><u>Ovaries<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Embryology<\/li>\n<li>Histology<\/li>\n<li>Anatomy<\/li>\n<li>Ovarian physiology<\/li>\n<li>Hypothalamic-pituitary-ovarian axis<\/li>\n<li>Oogenesis<\/li>\n<li>Peripheral actions of estrogen and progesterone<\/li>\n<li>Mechanism of action of ovarian hormones<\/li>\n<\/ul>\n<ol start=\"11\">\n<li><strong><u>Normal puberty<\/u><\/strong><\/li>\n<li><strong><u>Influence of the endocrine glands on development and growth<\/u><\/strong>\n<ul>\n<li>Growth hormone<\/li>\n<li>Thyroid hormones<\/li>\n<li>Insulin<\/li>\n<li>Glucocorticoids<\/li>\n<li>Androgens<\/li>\n<li>Estrogen<\/li>\n<li>Progesterone<\/li>\n<li>Peptide growth factors<\/li>\n<li>Control of growth at the cellular level<\/li>\n<li>Prenatal growth<\/li>\n<li>Growth from birth to puberty<\/li>\n<li>Pubertal growth<\/li>\n<li>Bone age and physical maturity<\/li>\n<li>Development of specific organs and tissues<\/li>\n<li>Growth by hyperplasia and growth by hypertrophy<\/li>\n<\/ul>\n<\/li>\n<\/ol>\n<p>&nbsp;<\/p>\n<ol start=\"13\">\n<li><strong><u>Normal sexual differentiation<\/u><\/strong>\n<ul>\n<li>Chromosomal sex and biological functions of the Y and X chromosomes<\/li>\n<li>H-Y antigen<\/li>\n<li>Differentiation of the gonads<\/li>\n<li>Differentiation of the internal genitals<\/li>\n<li>Differentiation of the external genitalia and urogenital sinus<\/li>\n<li>Sexual differentiation in the hypothalamus<\/li>\n<li>Psycho-sexual differentiation<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Genetics and endocrinology<\/u><\/strong>\n<ul>\n<li>Mitosis and meiosis<\/li>\n<li>Normal human chromosomes<\/li>\n<li>Chromosomal aberrations<\/li>\n<\/ul>\n<\/li>\n<\/ol>\n<ul>\n<li>Structural<\/li>\n<li>Numerical\n<ul>\n<li>Interpretation<\/li>\n<li>Interpretation<\/li>\n<li>Chromosomal alterations of the most common endocrine diseases<\/li>\n<li>General principles of hereditary diseases<\/li>\n<li>Genetic action endocrine glands<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<ol start=\"15\">\n<li><strong><u>Immunology<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>General concepts<\/li>\n<li>Concept of antigens<\/li>\n<li>Concept of antibodies<\/li>\n<li>Types of immunity (autoimmunity, humoral immunity and cellular immunity)<\/li>\n<li>Concept of HLA and its importance in pathology<\/li>\n<li>Autoimmunity and endocrine diseases<\/li>\n<li>Mechanisms involved in the production of autoimmune endocrinopathies<\/li>\n<li>Predisposing factors: environmental and genetic<\/li>\n<li>Autoimmune polyendocrine syndrome<\/li>\n<li>Nature of the immune response<\/li>\n<li>Immune-mediated tissue injury<\/li>\n<li>Influence of the endocrine system in the expression of allergic and immunological reactions<\/li>\n<\/ul>\n<ul>\n<li>Allergic and immunological reactions to hormones<\/li>\n<\/ul>\n<ol start=\"16\">\n<li><strong>Skin and hormones<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Hormones, the epidermis and dermis<\/li>\n<li>Dermal vascularization and hormones<\/li>\n<li>Hormones, melanocytes and pigmentation<\/li>\n<\/ul>\n<ol>\n<li><strong><u>Hypopituitarism<\/u><\/strong>\n<ul>\n<li>Concept of panhypopituitarism and selective and partial hypopituitarism<\/li>\n<li>Causal classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis (anorexia nervosa, primary hypothyroidism primary adrenal insufficiency, primary hypogonadism, anemias, hypoglycemic syndrome, cachexia from other causes, short stature from other causes)<\/li>\n<\/ul>\n<\/li>\n<li>Study program<\/li>\n<li><strong><u>Acromegaly and gigantism<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Etiology<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Prolactinomas and hyperprolactinemia<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Frequency<\/li>\n<li>Clinical picture<\/li>\n<\/ul>\n<\/li>\n<\/ol>\n<p>Differential diagnosis<\/p>\n<ol start=\"4\">\n<li><strong><u>Other pituitary tumors<\/u><\/strong>\n<ul>\n<li>Concept of pituitary tumor<\/li>\n<li>Concept of macro and micro adenoma<\/li>\n<li>Variants of tumor growth<\/li>\n<li>Classification according to hormonal secretion vs staining classification<\/li>\n<li>Clinical picture<\/li>\n<li>Other non-pituitary tumors (craniopharyngioma)<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Mild polyureic-polydipsic syndrome<\/u><\/strong>\n<ul>\n<li>Concept of VSDI, VRDI and potomania<\/li>\n<li>Pathogenic classification<\/li>\n<li>Clinical picture<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Short stature<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Etiological diagnosis<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<li>Bone age determination, oral chromatin and skull radiology in the study of short stature syndrome<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Tall stature syndrome<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Etiological diagnosis<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Hyperthyroidism<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Classification<\/li>\n<li>Clinical picture varieties<\/li>\n<li>Differential diagnosis<\/li>\n<li>Etiopathogenesis of Graves-Basedow disease<\/li>\n<li>Clinical manifestations of Graves-Basedow disease<\/li>\n<li>Thyrotoxic crisis diagnosis and treatment<\/li>\n<li>Hyperthyroidism study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Thyroiditis<\/u><\/strong>\n<ul>\n<li>Acute thyroiditis, etiology, diagnosis<\/li>\n<li>Subacute thyroiditis, etiology, diagnosis<\/li>\n<li>Hashimoto\u2019s disease, etiology, diagnosis<\/li>\n<li>Chronic, mycotic and bacterial thyroiditis, etiology, diagnosis<\/li>\n<li>Riedel&#8217;s thyroiditis, etiology, diagnosis<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Hypothyroidism<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<li>Hypothyroidism in children<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Single thyroid nodule, nodular goiter, and thyroid cancer<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Clinical diagnosis<\/li>\n<li>Pathological classification of the thyroid nodule<\/li>\n<li>Study program<\/li>\n<li>Clinical manifestation and survival of thyroid cancer<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Genetic alterations that affect the synthesis, transport and utilization of thyroid hormones<\/u><\/strong>\n<ul>\n<li>Intrathyroidal enzyme abnormalities, diagnosis and treatment<\/li>\n<li>Uptake defect<\/li>\n<li>Organification defect<\/li>\n<li>Proteolysis defect<\/li>\n<li>Coupling defect<\/li>\n<li>Dehalogenation defect\n<ul>\n<li>Extrathyroidal genetic alterations, diagnosis and treatment<\/li>\n<\/ul>\n<\/li>\n<li>Changes in TBG serum concentration<\/li>\n<li>Peripheral resistance to thyroid hormones<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Euthyroid goiter<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Clinical diagnosis and etiology<\/li>\n<li>Differential diagnosis<\/li>\n<li>Complementary examinations<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Hyperparathyroidism<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concepts of hypercalcemia and primary, secondary and tertiary hyperparathyroidism<\/li>\n<li>Etiology<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis\n<ul>\n<li>Study program<\/li>\n<li>Other causes of hypercalcemia<\/li>\n<li>Examinations that aid in the localization of a tumor<\/li>\n<\/ul>\n<\/li>\n<\/ul>\n<ol start=\"15\">\n<li><strong><u>Hypoparathyroidism<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept<\/li>\n<li>Etiological classification\n<ol>\n<li>Causes with inadequate secretion of PTH<\/li>\n<li>Causes in which the secretion is normal but biologically ineffective<\/li>\n<li>Peripheral resistance to PTH (pseudohypoparathyroidism)<\/li>\n<\/ol>\n<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<li>Other causes of hypocalcemia<\/li>\n<li>Pseudo-pseudohypoparathyroidism<\/li>\n<li>Rickets and osteomalacia\n<ol>\n<li><strong><u>Diabetes mellitus (DM)<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Definition<\/li>\n<li>Classification<\/li>\n<li>Conditions and associated syndromes<\/li>\n<li>Impaired glucose tolerance (IGT), previous and potential<\/li>\n<li>Pathophysiology<\/li>\n<li>Clinical picture<\/li>\n<\/ul>\n<ol start=\"17\">\n<li><strong> <u> Etiopathogenesis of type 1 DM<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Environmental factors<\/li>\n<li>Immunological factors<\/li>\n<li>Inherited genetic factors<\/li>\n<li>Role of the HLA system in the etiopathogenesis of type 1 DM<\/li>\n<li>Insulin gene polymorphism<\/li>\n<\/ul>\n<ol start=\"18\">\n<li><strong><u>Etiopathogenesis of type 2 DM<\/u><\/strong><\/li>\n<li><strong><u>Diabetic ketoacidosis<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept of ketosis and ketoacidosis<\/li>\n<li>Triggers<\/li>\n<li>Clinical picture<\/li>\n<li>Complementary examinations<\/li>\n<li>Differential diagnosis\n<ol>\n<li><strong><u>Hyperosmolar coma and lactic acidosis<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Clinical picture<\/li>\n<li>Triggers<\/li>\n<li>Differential diagnosis<\/li>\n<li>Complementary examinations<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<ol start=\"21\">\n<li><strong><u>DM in children and adolescents<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Diagnosis<\/li>\n<li>Control criteria<\/li>\n<li>Acute complications\n<ol>\n<li><strong><u>Diabetes and pregnancy<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Gestational diabetes, concept and diagnosis<\/li>\n<li>Potential abnormality of glucose tolerance<\/li>\n<li>Most frequent congenital malformations in the child of a diabetic mother\n<ol>\n<li><strong><u>Ocular complications of DM Diagnosis<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Retina<\/li>\n<li>Iris<\/li>\n<li>Crystalline<\/li>\n<li>Conjunctiva<\/li>\n<li>Cornea<\/li>\n<li>Intraocular muscles<\/li>\n<li>Nerves<\/li>\n<\/ul>\n<ol start=\"24\">\n<li><strong><u>Diabetic nephropathy<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept<\/li>\n<li>Histological classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Complementary examinations<\/li>\n<\/ul>\n<ol start=\"25\">\n<li><strong><u>Diabetic neuropathy<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept<\/li>\n<li>Classification and diagnosis<\/li>\n<li>Complementary examinations<\/li>\n<\/ul>\n<ol start=\"26\">\n<li><strong><u>Skeletal and joint complications of DM<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Neuropathic arthropathy<\/li>\n<li>Diabetic osteopathy<\/li>\n<li>Osteopenia<\/li>\n<li>Articular syndromes of the hand<\/li>\n<\/ul>\n<ol start=\"27\">\n<li><strong><u>Vascular complications of DM<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Macroangiopathy<\/li>\n<li>Microangiopathy<\/li>\n<\/ul>\n<ol start=\"28\">\n<li><strong><u>Dermatology complications of DM<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Ochre dermatitis<\/li>\n<li>Diabetic dermopathy<\/li>\n<li>Necrobiosis lipoidica diabeticorum<\/li>\n<li>Diabetic bullae<\/li>\n<li>Granuloma annulare\n<ol>\n<li><strong><u>Diabetic foot<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Etiopathogenesis<\/li>\n<li>Classification<\/li>\n<li>Clinical picture<\/li>\n<li>Complementary examinations<\/li>\n<\/ul>\n<ol start=\"30\">\n<li><strong><u>Special situations in diabetic treatment<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Hypoglycemia<\/li>\n<li>Insulin lipodystrophy<\/li>\n<li>Insulin allergy<\/li>\n<li>Insulin resistance<\/li>\n<li>Other diabetic comas<\/li>\n<li>Infections<\/li>\n<li>Malignant otitis<\/li>\n<li>Acute hepatitis<\/li>\n<li>Hyperthyroidism<\/li>\n<li>Acute myocardial infarction (AMI)\n<ol>\n<li><strong><u>Hypoglycemic syndrome<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program\n<ol>\n<li><strong><u>Hyperlipoproteinemia<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concepts of hyperlipidemia, hyperlipemia, \u03b1-lipoproteins, \u03b2-lipoproteins, pre beta lipoproteins, wide \u03b2-lipoprotein<\/li>\n<li>Mechanism of absorption and digestion of lipids<\/li>\n<li>Metabolism of lipoproteins<\/li>\n<li>Concept and types of apoproteins, function and properties<\/li>\n<li>Classification of hyperlipoproteinemia<\/li>\n<li>Diagnosis<\/li>\n<\/ul>\n<ol start=\"33\">\n<li><strong><u>Obesity<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Diagnosis<\/li>\n<li>Differential diagnosis<\/li>\n<li>Classification<\/li>\n<li>Complications<\/li>\n<li>Study program\n<ol start=\"34\">\n<li><strong><u>Cushing\u2019s disease<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Adrenal insufficiency<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept<\/li>\n<li>Clinical picture and differential diagnosis<\/li>\n<li>Causal classification<\/li>\n<li>Study program\n<ol>\n<li><strong><u>Congenital adrenal hyperplasia (CAH)<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Enzyme defects that produce congenital adrenal hyperplasia<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<li><\/li>\n<\/ul>\n<ol start=\"37\">\n<li><strong><u>Primary hyperaldosteronism<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Pheochromocytoma<\/u><\/strong>\n<ul>\n<li>Concept<\/li>\n<li>Classification<\/li>\n<li>Clinical picture<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program\n<ol start=\"39\">\n<li><strong><u>Amenorrhea<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept of primary and secondary amenorrhea<\/li>\n<li>Concept of oligomenorrhea<\/li>\n<li>Causal classification<\/li>\n<li>Etiological diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<\/li>\n<li><strong><u>Female hypogonadism<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Causal classification<\/li>\n<li>Concept of primary and secondary hypogonadism<\/li>\n<li>Clinical picture<\/li>\n<li>Study program\n<ol>\n<li><strong><u>Abnormal uterine bleeding<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Causal classification<\/li>\n<li>Etiological diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<ol start=\"42\">\n<li><strong><u>Hirsutism<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept of hirsutism and hypertrichosis<\/li>\n<li>Assessment of the severity of hirsutism, quantitative index<\/li>\n<li>Causal classification<\/li>\n<li>Etiological diagnosis<\/li>\n<li>Study program\n<ol>\n<li><strong><u>Infertility (masculine and feminine)<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concepts<\/li>\n<li>Causal classification<\/li>\n<li>Diagnosis<\/li>\n<li>Study program\n<ol>\n<li><strong><u>Masculine hypogonadism<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Causal classification<\/li>\n<li>Concept of primary and secondary, pre- and post-pubertal hypogonadism<\/li>\n<li>Clinical picture<\/li>\n<li>Study program<\/li>\n<\/ul>\n<ol start=\"45\">\n<li><strong><u>Empty scrotum syndrome<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept of cryptorchidism, retractile testicles, testicular ectopia and anorchia<\/li>\n<li>Clinical diagnosis<\/li>\n<li>Complementary examinations\n<ol>\n<li><strong><u>Pubertal delay<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept<\/li>\n<li>Clinical diagnosis and etiology<\/li>\n<li>Study program<\/li>\n<\/ul>\n<ol start=\"47\">\n<li><strong><u>Variations in pubertal development<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Premature pubarche or adrenarche<\/li>\n<li>Premature thelarche<\/li>\n<li>Premature menarche<\/li>\n<li>Virginal breast hypertrophy<\/li>\n<li>Small breasts<\/li>\n<li>Gynecomastia<\/li>\n<li>Menstrual disorders\n<ol>\n<li><strong><u>Early sexual development<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Concept of true early puberty and sexual precocity<\/li>\n<li>Etiological classification<\/li>\n<li>Clinical diagnosis and etiology<\/li>\n<li>Differential diagnosis<\/li>\n<li>Study program<\/li>\n<\/ul>\n<ol start=\"49\">\n<li><strong><u>Hypogenitalism<\/u><\/strong><\/li>\n<\/ol>\n<ul>\n<li>Concept<\/li>\n<li>Classification<\/li>\n<li>Clinical diagnosis<\/li>\n<li>Study program\n<ol>\n<li><strong><u>Abnormalities of sexual differentiation<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<\/ul>\n<ul>\n<li>Concept<\/li>\n<li>Classification<\/li>\n<li>Concept of ambiguous genitalia<\/li>\n<li>Differential diagnosis of sexual differentiation abnormalities<\/li>\n<li>Gonadal dysgenesis: Turner, mixed and pure; diagnosis and treatment<\/li>\n<li>Klinefelter syndrome Diagnosis and treatment<\/li>\n<li>Masculine and feminine pseudo hermaphroditism, diagnosis<\/li>\n<\/ul>\n<ol start=\"51\">\n<li><strong><u>Gonadal tumors with endocrine function<\/u><\/strong>\n<ul>\n<li>Ovarian tumors<\/li>\n<li>Testicular tumors<\/li>\n<li>Classification<\/li>\n<li>Diagnosis\n<ol start=\"52\">\n<li><strong><u>Systemic contraception<\/u><\/strong><\/li>\n<li><strong>Multiple endocrine adenomatosis (MEN)<\/strong><\/li>\n<li><strong><u>Endocrine hypertension<\/u><\/strong><\/li>\n<\/ol>\n<\/li>\n<li>Renin-aldosterone axis in blood pressure control<\/li>\n<li>Renin system description<\/li>\n<li>Abnormalities of aldosterone and renin secretion<\/li>\n<li>Electrolyte balance and hypertension diagnosis<\/li>\n<\/ul>\n<\/li>\n<\/ol>\n<p>&nbsp;<\/p>\n<ol>\n<li><strong>Hypopituitarism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"2\">\n<li><strong>Acromegaly and gigantism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"3\">\n<li><strong>Prolactinomas and hyperprolactinemia<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"4\">\n<li><strong>Other pituitary tumors<\/strong><\/li>\n<li><strong>Mild polyuria-polydipsia syndrome<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"6\">\n<li><strong>Therapeutic approach to tall stature syndrome<\/strong><\/li>\n<li><strong>Therapeutic approach to short stature syndrome<\/strong><\/li>\n<li><strong>Hyperthyroidism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Treatment with medicine, surgery and I-131<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"9\">\n<li><strong>Thyroiditis<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"10\">\n<li><strong>Hypothyroidism:<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"11\">\n<li><strong>Single thyroid nodule, nodular goiter, and thyroid cancer<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Indications of surgical treatment<\/li>\n<li>Other forms of treatment<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"12\">\n<li><strong>Euthyroid goiter<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"13\">\n<li><strong>Hyperparathyroidism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Indications of surgical treatment<\/li>\n<li>Other forms of treatment<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"14\">\n<li><strong>Hypoparathyroidism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"15\">\n<li><strong>Diabetes mellitus (DM) and AGT<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of treatment<\/li>\n<li>Preventative treatment<\/li>\n<li>Diabetes education<\/li>\n<li>Diet<\/li>\n<li>Exercises<\/li>\n<li>Oral hypoglycemia, indications and contraindications, drugs used and mechanisms of action<\/li>\n<li>Metabolic control criteria<\/li>\n<\/ul>\n<ol start=\"16\">\n<li><strong>Diabetic ketoacidosis<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"17\">\n<li><strong>Hyperosmolar coma and lactic acidosis<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"18\">\n<li><strong>DM in children and adolescents<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Dosages and routes of administration<\/li>\n<li>Other forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Metabolic control criteria<\/li>\n<li>Treatment for acute complications of DM<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"19\">\n<li><strong>Diabetes and pregnancy<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Diets for pregnant women<\/li>\n<li>Insulin treatment schemes<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"20\">\n<li><strong>Ocular complications of DM<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"21\">\n<li><strong>Diabetic nephropathy<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"22\">\n<li><strong>Diabetic neuropathy<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"23\">\n<li><strong>Skeletal and joint complications of DM<\/strong><\/li>\n<li><strong>Diabetic foot<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"25\">\n<li><strong>Special situations in diabetic treatment<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Hypoglycemia<\/li>\n<li>Insulin lipodystrophy<\/li>\n<li>Insulin allergy<\/li>\n<li>Insulin resistance<\/li>\n<li>Other diabetic comas<\/li>\n<li>Infections<\/li>\n<li>Malignant otitis<\/li>\n<li>Acute hepatitis<\/li>\n<li>Hyperthyroidism<\/li>\n<li>Acute myocardial infarction (AMI)<\/li>\n<\/ul>\n<ol start=\"26\">\n<li><strong>Hypoglycemic syndrome<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"27\">\n<li><strong>Hyperlipoproteinemia<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Diets<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"28\">\n<li><strong>Obesity<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment.<\/li>\n<li>Forms of treatment<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"29\">\n<li><strong>Cushing\u2019s disease<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Surgical criteria<\/li>\n<li>Pre-surgical preparation<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"30\">\n<li><strong>Adrenal insufficiency<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"31\">\n<li><strong>Congenital adrenal hyperplasia (CAH)<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"32\">\n<li><strong>Primary hyperaldosteronism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"33\">\n<li><strong>Pheochromocytoma<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Pre-surgical preparation<\/li>\n<li>Most common complications<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"34\">\n<li><strong>Treatment for types of Amenorrhea<\/strong><\/li>\n<li><strong>Female hypogonadism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<\/ul>\n<ol start=\"36\">\n<li><strong>Treatment of abnormal uterine bleeding<\/strong><\/li>\n<li><strong>Hirsutism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Secondary effects<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<ol start=\"38\">\n<li><strong>Therapeutic approach to infertility treatment (masculine and feminine)<\/strong><\/li>\n<li><strong>Masculine hypogonadism<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<\/ul>\n<ol start=\"40\">\n<li><strong>Therapeutic approach to empty scrotum syndrome<\/strong><\/li>\n<li><strong>Therapeutic approach to pubertal delay<\/strong><\/li>\n<li><strong>Physical variations in pubertal development<\/strong><\/li>\n<li><strong>Therapeutic approach to early sexual development<\/strong><\/li>\n<li><strong>Therapeutic approach to hypogenitalism<\/strong><\/li>\n<li><strong>Therapeutic approach to sexual differentiation abnormalities<\/strong><\/li>\n<li><strong>Therapeutic approach to gonadal tumors with endocrine function<\/strong><\/li>\n<li><strong>Systemic contraception<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Types of contraceptives<\/li>\n<li>Routes of administration<\/li>\n<li>Treatment schemes<\/li>\n<li>Secondary effects<\/li>\n<\/ul>\n<ol start=\"48\">\n<li><strong>Therapeutic approach to multiple endocrine adenomatosis (MEN)<\/strong><\/li>\n<li><strong>Endocrine hypertension<\/strong><\/li>\n<\/ol>\n<ul>\n<li>Objectives of comprehensive treatment<\/li>\n<li>Forms of treatment<\/li>\n<li>Evolution<\/li>\n<li>Prognosis<\/li>\n<\/ul>\n<p>&nbsp;<\/p>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>YEAR RESIDENCY MONTHS WEEKS HOURS &nbsp; First Internal medicine &nbsp; Endocrinology service for adults 3* &nbsp; 8 13 &nbsp; 35 60 hours per week** &nbsp; 60 hours per week &nbsp; &nbsp; Second General endocrinology Endocrinology and pregnancy &nbsp; Center for education and care of the diabetic patient Endocrinology laboratory 6 2 &nbsp; 1 2 24<\/p>\n","protected":false},"featured_media":763127,"template":"","meta":{"_monsterinsights_skip_tracking":false,"_monsterinsights_sitenote_active":false,"_monsterinsights_sitenote_note":"","_monsterinsights_sitenote_category":0},"product_brand":[],"product_cat":[231],"product_tag":[],"class_list":{"0":"post-760426","1":"product","2":"type-product","3":"status-publish","4":"has-post-thumbnail","6":"product_cat-programas-de-formacion-especialidades-medicas-en","8":"first","9":"instock","10":"virtual","11":"purchasable","12":"product-type-simple"},"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.3 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Endocrinology Specialization Training Program (For European Resident Physicians - Gestoria Pyrenees WT<\/title>\n<meta name=\"robots\" content=\"index, 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